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FARAFARA Cure FA

Protective effects of transduced PEP-1-Frataxin protein on oxidative stress-induced neuronal cell death

Reactive oxygen species (ROS) actively contribute to the development of a number of human diseases including ischemia. In response to oxidative stress, frataxin has a significant ability to improve cell survival though its biological function is unclear in relation to ischemia. To explore frataxin's role in protecting against ischemic cell death, we constructed PEP-1-Frataxin cell-permeable fusion protein.

Protective effects of transduced PEP-1-Frataxin protein on oxidative stress-induced neuronal cell death

About the Author

Jen Farmer

Jen Farmer

Executive Director

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